I assume you mean G6PD *deficiency*? I hope not, but maybe it could stay undiagnosed in some cases? "Glucose-6-phosphate dehydrogenase (G6PD) is an enzyme in the pentose phosphate pathway (see image), a metabolic pathway that supplies reducing energy to cells (most notably erythrocytes) by maintaining the level of the co-enzyme nicotinamide adenine dinucleotide phosphate (NADPH). The NADPH in turn maintains the level of glutathione in these cells that helps protect the red blood cells against oxidative damage. G6PD converts glucose-6-phosphate into 6-phosphoglucono-δ-lactone and is the rate-limiting enzyme of the pentose phosphate pathway." "Glucose-6-phosphate dehydrogenase deficiency is very common worldwide, and cause acute hemolytic anemia in the presence of simple infection, ingestion of fava beans, or reaction with certain medicines, AAA, antibiotics, antipyretics, and antimalarials." Source and further information: http://en.wikipedia.org/wiki/G6PD 2) "Patients are almost exclusively male, due to the X-linked pattern of inheritance, but female carriers can be clinically affected due to lyonization where random inactivation of an X-chromosome in certain cells creates a population of G6PD deficient red cells coexisting with normal red cells. G6PD manifests itself in a number of ways: Prolonged neonatal jaundice Hemolytic crises in response to: Certain drugs (see below) Certain foods, most notably broad beans Illness (severe infections) Diabetic ketoacidosis Very severe crises can cause acute renal failure Favism is a disorder characterized by hemolytic anemia in response to ingestion of fava beans." "The diagnosis is generally suspected when patients from certain ethnic groups (see below) develop anemia, jaundice and symptoms of hemolysis after challenge to any of the above causes, especially when there is a positive family history." Source and further information: http://en.wikipedia.org/wiki/Glucose-6-phosphate_dehydrogenase_deficiency 3) do you have it and how do you deal with it?